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Wuhan Dahua Weiye Pharmaceutical Chemical Co., Ltd.

Swimming, Stearoyl Vanillylamide, Gw501516 manufacturer / supplier in China, offering China Supply Increase Swimming Capacity Stearoyl Vanillylamide, Reduce Blood Press Losartan Potassium for Hypertension Treatment (CAS 124750-99-8), Injectable Anabolic Steroids Supplements Trenbolone Acetate and so on.

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Supplier Homepage Product Sarms Powder China Supply Increase Swimming Capacity Stearoyl Vanillylamide

China Supply Increase Swimming Capacity Stearoyl Vanillylamide

FOB Price: US $1 / kg
Min. Order: 200 kg
Min. Order FOB Price
200 kg US $1/ kg
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Production Capacity: 5000kg/Month
Transport Package: According to Customer′s Requirements
Payment Terms: T/T, Western Union, Paypal, Money Gram, Bitcoin

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Basic Info
  • Model NO.: CAS#: 58493-50-8
  • Customized: Non-Customized
  • Suitable for: Adult
  • Purity: >99%
  • Molecular Formula:  C14h15cl2n3OS
  • Specification: USP/BP, EP
  • HS Code: 3822009000
  • Powder: Yes
  • Certification: GMP, USP
  • State: Sarms Powder
  • Molecular Weight: : 344.256
  • Trademark: Dahua
  • Origin: China
Product Description

Dear friend, thanks for visiting our website. This is Luna, I can check email and reply you at any time. Any items that you are interested in, pls feel free to contact me, I will reply you at the first time, and I will value every chance to be your reliable partner.

What is Stearoyl Vanillylamide ?
Stearoyl Vanillylamide is an analog of Capsaicin, a compound found in various species of Hot Pepper which is primarily responsible for the spiciness. Unlike Capsaicin, however, Stearoyl Vanillylamide is non-pungent, which actually makes it easier to use in powder-form supplements.
How Stearoyl Vanillylamide works
Stearoyl Vanillylamide has been shown to increase swimming capacity in mice, compared to mice given a placebo.
According studies: Intravenous injection of stearoyl vanillylamide (C18-VA), a nonpungent capsaicin (CAP) analog,enhances adrenaline secretion significantly and as effectively as CAP in rats. Because swimming capacity was enhanced by CAP in mice due to CAP-induced adrenal catecholamine secretion, we investigated the effects of oral administration of C18-VA on swimming capacity using an adjustable-current water pool. Male Std ddY 6-wk-old mice were fed a commercial diet for this study and one group was orally administered C18-VA via a stomach tube. Treated mice were able to swim longer before exhaustion than the control mice (62.9 6 5.6 vs. 49.6 6 7.0 min, P , 0.05). The swimming capacity of two groups administered C18-VA (0.02 and 0.033 mmol/kg) was significantly greater than that of those administered vehicle alone, (P , 0.05). Substance P concentration in cerebrospinal fluid, which is involved in pain transmission and is the first direct measure of pungency, was not affected by C18-VA administration. In an experiment examining the effects of C18-VA on serum adrenaline concentration, adrenaline was significantly greater in C18-VA treated mice than in controls at 2-h post-dose (C18-VA group, 26.09 6 2.82; control group 13.29 6 0.96 mg/L, P , 0.01). In a separate study free fatty acids in serum were elevated in treated mice at 2-h post-dose (P , 0.01). While serum glucose concentration was not affected. These results suggest that C18-VA increased swimming capacity of mice via adrenaline release, independent of pungency.

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